Fig. 1From: Diabetes as a potential compounding factor in COVID-19-mediated male subfertilityDirect infection of testicular cells by SARS-CoV-2. By binding to the highly expressed viral entry receptors on testicular cells, specifically the Sertoli cells, Leydig cells, and spermatogonia, SARS-CoV-2 may infect these cells and cause direct cell damage, metabolic reprogramming, and/or immune responses. The result would be cell dysfunction and apoptosisBack to article page