Fig. 6From: Current insights into the implications of m6A RNA methylation and autophagy interaction in human diseasesEffects of m6A-autophagy interaction in cancer drug resistance. A NSCLC could develop gefitinib resistance through m6A mediated activation of autophagy. B HCC could develop sorafenib resistance through m6A mediated decreased expression of FOXO3, which contributes to activation of autophagy. C Defective autophagy and m6A mediated increased stability of ARHGAP5 transcripts could promote chemoresistance in gastric cancer cellsBack to article page