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Table 3 KEGG 2019 and Reactome 2016 resulting pathways that were common between the three tissues

From: Transcriptomic characterization of tissues from patients and subsequent pathway analyses reveal biological pathways that are implicated in spastic ataxia

Database iPSC-derived neurons and FCLs and LCLs iPSC-derived neurons and FCLs iPSC-derived neurons and LCLs FCLs and LCLs
KEGG 2019 Proteoglycans in cancer Melanoma   Salmonella infection
  C-type lectin receptor signaling pathway
  Chagas disease (American trypanosomiasis)
  Hematopoietic cell lineage
  Cytokine-cytokine receptor interaction
   Axon guidance
Reactome 2016 Cytokine signaling in immune system Defective CHST6 causes MCDC1 Interferon alpha/beta signaling Termination of translesion DNA synthesis
Defective ST3GAL3 causes MCT12 and EIEE15 DNA Damage Bypass
Defective B4GALT1 causes B4GALT1-CDG (CDG-2d) Gap-filling DNA repair synthesis and ligation in GG-NER
Synthesis of Prostaglandins (PG) and Thromboxanes (TX) Growth hormone receptor signaling
Non-integrin membrane-ECM interactions Nicotinate metabolism
Keratan sulfate degradation Signaling by Interleukins
Extracellular matrix organization Recognition of DNA damage by PCNA-containing replication complex
Diseases associated with glycosaminoglycan metabolism Immune system Rho GTPase cycle
HS-GAG biosynthesis  
Glycosaminoglycan metabolism Senescence-Associated Secretory Phenotype (SASP)
Heparan sulfate/heparin (HS-GAG) metabolism  
ECM proteoglycans Axon guidance
Cell surface interactions at the vascular wall