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Fig. 6 | Cell & Bioscience

Fig. 6

From: Role of ammonia for brain abnormal protein glycosylation during the development of hepatitis B virus-related liver diseases

Fig. 6

Calcium activity in the brain of HBV transgenic mice and a molecular network. a The protein expression levels of C1GALT1 and IP3R1 in the brains of 18 months old HBV transgenic mice and control mice. b Ca2+ concentration in the brains of 18 months old HBV transgenic mice and control mice. c A proposed molecular network, using lectin microarrays approach to assess glycosylation in brains of HBV transgenic mice and human astrocytes, find increased Galβ1-3GalNAc mediated by C1GALT1 in hyperammonemia and C1GALT1 facilitates calcium concentration due to the overexpression of IP3R1

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