Fig. 1From: Role of ammonia for brain abnormal protein glycosylation during the development of hepatitis B virus-related liver diseasesHepatic injury, blood ammonia concentration and astrocytes in HBV transgenic mice. a The H&E staining of mice liver tissues at 4 to 18 months old. Fatty liver was marked with a black box, and liver fibrosis was marked with white a box. Scale bar, 200/500 μm. b The serum ammonia concentration of HBV transgenic mice and control mice at 4 to 18 months old. Each group of serum came from a mixture of six mice, at least three replicates per condition. c The H&E staining of mice cerebral hemisphere and midbrain at 10 to 18 months old. Scale bar, 50 μm. d–f, The diameter (1000×) of astrocytes in the left hemispheres (d), right hemispheres (e) and midbrain (f)Back to article page